Osteopontin is indispensible for AP1-mediated angiotensin II-related miR-21 transcription during cardiac fibrosis
Author(s)
Type
Journal Article
Date Issued
2015-04-21
Date Acceptance
2015-03-17
Citation
European Heart Journal, 2015, 36 (32), pp.2184-2196
ISSN
1522-9645
Publisher
Oxford University Press
Start Page
2184
End Page
2196
Journal / Book Title
European Heart Journal
Volume
36
Issue
32
Copyright Statement
This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits non-commercial re-use, distribution, and reproduction in any medium, provided the original work is properly cited.
Subjects
Science & Technology
Life Sciences & Biomedicine
Cardiac & Cardiovascular Systems
Cardiovascular System & Cardiology
Osteopontin
Cardiac fibrosis
microRNA
Angiotensin II
miR-21
PROMATRIX METALLOPROTEINASE-9 ACTIVATION
CHRONIC KIDNEY-DISEASE
SMOOTH-MUSCLE-CELLS
ESSENTIAL-HYPERTENSION
HEART-FAILURE
MESSENGER-RNA
EXPRESSION
INFLAMMATION
MECHANISM
BLOCKER
Adenoviridae
Aged
Animals
Cell Survival
Cells, Cultured
Collagen
Female
Fibrosis
Gene Silencing
Genetic Vectors
Humans
In Vitro Techniques
Male
Mice, Knockout
MicroRNAs
Myocardium
Myofibroblasts
PTEN Phosphohydrolase
Phosphatidylinositol 3-Kinases
Recombinant Proteins
Transcription Factors
Cardiovascular System & Hematology
1102 Cardiovascular Medicine And Haematology
Publication Status
Published